Ciliary neurotrophic factor protects nerve cells Since its discovery as a proteins that gets specifically released in response to brain injury, ciliary neurotrophic factor has prompted much interest as a potential therapeutic agent link . However, many experiments have fulfilled with limited success, until now; a research team shows that co-administrating CNTF with its receptor promotes the growth and survival of neurons. Related StoriesUnderstanding how schizophrenia affects workings of the brainPresence of connexin proteins suppresses primary tumor growthNew Global Energy and International Sustainability Group agree to manufacture, distribute MoringaUP Protein BarsWhile the receptor for CNTF is generally tied to the top of neurons, this tether is frequently cut off during trauma, which led Tag Ozog, Christian colleagues and Naus to suspect that CNTF and the free-floating receptor might act in a complex.
Peter Shang-Tzen Chang, a professor in the institution of Forestry and Source Conservation at the National Taiwan University tested eleven substances in cinnamon leaf oil for their capability to eliminate emerging larvae of the yellow fever mosquito, Aedes aegypti. Aegypti in a day of testing,’ Chang says. Larvicidal activity is definitely judged with a measurement known as LC50. ‘The LC50 value is the concentration that kills 50 % of mosquito larvae in a day,’ Chang explains. Decrease LC50 translates into higher activity, because it takes a lower concentration to eliminate larvae in the same amount of time. All compounds had LC50 values of significantly less than 50 parts per million , with cinnamaldehyde displaying the strongest activity at an LC50 of 29 ppm.